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Whether GSH-dependent copper trafficking and Fe-S cluster maintenance actively license ferroptosis or cuproptosis, rather than simply buffering stress, remains unclear

Owing to its high concentration in the cell (~10 3 M) 1 , the GSH/GSSG pair acts as the main intracellular redox buffer, plays a key role in the detoxification of reactive oxygen species (ROS), and participates in redox signalling 2

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Cross-reference your calculation with the appropriate concentration table above

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