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TNF- damages cardiomyocyte mitochondria by inhibiting glycolysis and reducing mitochondrial adenosine triphosphate (ATP) production through promoting oxidative and nitrosative stress, which can result in compromised cardiac systolic function and unfavorable cardiac remodeling [83, 84]

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A 2006 study published in the Journal of Clinical Endocrinology and Metabolism found that CJC-1295 produced sustained, dose-dependent increases in both GH and IGF-1 levels