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When ET-1 is administered in high concentrations, it behaves as a potent vasoconstrictor that is capable of exerting an array of physiological effects, including the potential to alter arterial pressure

This faster regeneration process leads to a quicker return to full function and a reduced risk of complications

Additionally, DSIP attenuates ACTH and corticosterone release in response to stress, indicating an effect on the hypothalamic-pituitary-adrenal axis