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In addition to the GPX4-dependent mechanisms, several GPX4-independent pathways contribute to ferroptosis regulation, including the NAD(P)H/FSP1/CoQ10 axis, the GCH1/BH4/DHFR axis, squalene accumulation, and DHODH-mediated suppression of ferroptosis [88, 92]

Acetyl buffering: maintains CoA/acetyl-CoA balance, influencing glucosefat fuel sparing. Anti-oxidant & anti-inflammatory actions: down-regulation of CRP, TNF- and lipid peroxidation noted in several meta-analyses

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