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The peptide's mechanism involves disrupting the FOXO4-p53 interaction specifically in replication-stressed senescent chondrocytes, triggering selective apoptosis while preserving healthy proliferative cells needed for cartilage formation

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After 48 h, cells were fixed and then analyzed with fluorescence-activated cell sorting and GFP-positive cells were detected

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10.1016/j.ejphar.2010.09.075 44 MahajanS