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A more recent transcriptome study on the E15.5 brains of control and Nsd2 KO mice revealed that the loss of NSD2 caused dysregulation of genes related to synaptic transmission and formation (Kinoshita et al., 2024)

Back and co-workers [46], reported the catalytic cycle of di(3-hydroxy-propyl) selenide and acts an efficient catalyst for the reduction of t-BuOOH in the presence of BnSH

doi:10.1042/bj3490455

13 Regarding the application of GLP-1RAs in MASLD, current understanding suggests they can serve as additional pharmaceutical options in lifestyle management for patients with complications like obesity and T2DM