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Preclinical studies have used various neurotoxin-based models to mimic the oxidative stress and dopaminergic neurodegeneration observed in PD

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Metabolite sensing mechanisms and switches (such as O-GlcNAcylation, N-acylation, lactylation and methylation) have been discovered to dynamically regulate the epigenetic programming of particular lineages cell fates during fetoplacental development

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