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As we knew a priori about age differences in the distinct variables, we executed a regression model for each group

Taken together, our data indicate that the loss of GSH triggered by Gclc deletion induces a defective mitochondrial accumulation in FoB, which may lead to compensation through upregulation of glycolytic metabolism

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Stress, boredom, or sadness no longer automatically lead to food-seeking behavior because the medication recalibrates the brain's food-motivation system