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This augmentation occurs either directly through the stimulation of TNFR1 by TNF released from necrotic cells or indirectly through the recruitment and activation of leukocytes including macrophages, neutrophils, lymphocytes, and other proinflammatory cells that contribute to tissue injury (Linkermann et al., 2014

En tales condiciones, el glutatin es beneficioso para modular la apoptosis de las clulas T y los niveles de citocinas