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Deoxygenation-induced and Ca(2+) dependent phosphatidylserine externalisation in red blood cells from normal individuals and sickle cell patients

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Early research demonstrated that erythrocytes retained functional caspase-3 and caspase-8 [22, 23]

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Phosphatase and tensin homolog (PTEN) dephosphorylates PIP 3 back to PIP 2 , thereby regulating the pathway

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