There is a large body of preclinical studies that provide compelling evidence of a functional BTR signaling system in enteroendocrine cells, the effects of non-nutritive BTR agonists on enteroendocrine hormone secretion, and the potential for stimulating intestinal BTRs to suppress energy intake and reduce postprandial glycemic excursions (59, 66)
They act through different tissue layers and carry different downtime and cost profiles
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EBioMedicine 74 , 103739 (2021)
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