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Consequently, NF-B is released and translocates to the nucleus where it transactivates multiple target genes strongly promoting inflammation [22,23]

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Generally, for the low-risk chemical-exposure scenarios (except Crystalline and Amorphous silica occupational scenarios), the PoDs occurred at higher concentrations than the corresponding human exposure values, whereas for all the high-risk chemical-exposure scenarios, there was a clear overlap between the PoDs and lung deposited mass (benchmark inhaled materials) or maximum plasma concentration (systemically available benchmark drugs)