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Mitochondrial dysfunction plays a key role in oocyte ageing and the decline in germ cell function, and an in-depth study of its molecular mechanisms and intervention strategies is highly important for slowing oocyte ageing, increasing fertility, and improving the success rate of assisted reproduction techniques

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To test this hypothesis, we modified the DNA strand exchange protocol by pre-incubating ssDNA with RPA before the addition of RAD51 with or without an increasing concentration of PDS5B (Fig