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These alterations of metabolism appear to benefit cancer cells, aiding in tumor proliferation and survival, through not yet fully understood mechanisms and interactions [2]

extend as needed for ongoing management

et LABORIT G., (1955), mentionne ci-dessus et LAPICQUE L

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THERAPEUTIC INTERVENTIONS CLINICAL EXAMPLES AND THERAPEUTIC FORMS: N-acetylcysteine (NAC) Improving cellular glutathione status remains a clinical challenge, though interventions which enhance glutathione synthesis have for the most part been successful, most notably N-acetylcysteine (NAC)

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