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Funding This research was supported by the Shanghai Shen Kang Hospital Development Center (SHDC12019106 and SHDC12019X27), National Natural Science Foundation of China (82371656), Shanghai Clinical Research Center for Gynecological Diseases (22MC1940200), and Five-year Project Plan of the Whole Life Cycle Health Research Institute of Fudan University: Construction Project (DGF50107-037001)
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Protective mechanism of humanin against oxidative stress in aging-related cardiovascular diseases
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& Timlin, J
Furthermore, it suppresses oxidative stress by scavenging free radicals and enhancing the activity of endogenous antioxidant enzymes such as superoxide dismutase (SOD) and catalase (Morya et al., 2025)
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