Lurie Children's Hospital
It has long been known that growth differentiation factor 8 (GDF8, also known as myostatin), acting via the activin type II receptors ActRIIA/B in skeletal muscle, is a major negative regulator of muscle mass 7,8,9
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Multiple reports indicate that some men who meet standard GLP-1 prescribing criteria are declined through the Maximus screening process
We generated fully human antibodies to specifically block GDF8 (trevogrumab) and ActA (garetosmab), and showed that combining these blocking antibodies results in much greater increases in muscle than either of the antibodies individually, with dual blockade matching the muscle growth induced by broader ActRIIA/B pathway blockade in both mice and non-human primates (NHPs) 28,31
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