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Sati H, Carrara E, Savoldi A, et al

A molecular mechanism for ibuprofen-mediated RhoA inhibition in neurons

Gender-specific changes in energy metabolism and protein degradation as major pathways affected in livers of mice treated with ibuprofen

The alterations in neuronal circuits in autism may be reversible by re-expression of NLGN3 , as an NLGN3- knockout mice model of non-syndromic autism showed impaired heterosynaptic competition and altered mGluR-dependent synaptic plasticity [58]

There is some evidence suggesting it may interfere with the efficacy of certain chemotherapy drugs or other medications processed by the liver

In this task, mGluR5 receptor knock-out mice presented deficits in the acquisition and had impaired long-term retention [29]