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34 Unfortunately, we lacked systematic access to glycated haemoglobin values, which could have indicated that cases in our study population were with well-managed diabetes inadvertently selected leading to a rate of birth defects similar to the general population

in replete individuals extra B12 is largely excreted and does not enhance energy or performance [2]

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Taken together, these guidelines reinforce SGLT2i as the backbone of CKD management across diabetic and non-diabetic disease, with GLP-1 RAs positioned as additive therapy for appropriately selected patients, rather than as replacements

The GLP-1 receptor agonist revolution comes to nephrology

I have to stick myself with a needle