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Alterations in mitochondria-associated ER membranes, excessive production of reactive oxygen species, impaired autophagic flux, disturbed calcium transport, mitochondrial fragmentation, and activation of inflammatory pathways collectively drive progressive organellar dysfunction ( Within this framework, further consideration of AP through the lens of inter-organellar communication may offer an important conceptual advance

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Developing a blood cell-based diagnostic test for myalgic encephalomyelitis/chronic fatigue syndrome using peripheral blood mononuclear cells

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