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Enterovirus infection, particularly by CVB, is a strong candidate environmental trigger, with evidence that viral persistence, cell stress, and immune evasion mechanisms may accelerate or initiate autoimmune responses

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194,195 The activated PI3K catalyzes the conversion of membrane phospholipid Phosphatidylinositol-4,5-bisphosphate (PIP2) into Phosphatidylinositol-3,4,5-trisphosphate (PIP3)