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This reduction underscores the potential of targeting this intersection to simultaneously block cell death and the inflammatory storm, thereby offering a novel and combined intervention strategy for retinal degenerative diseases (141)

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a , Schematic diagram showing sequences of artificial microRNA (amiRNA) and targeted sites on Mp RALF1 mRNA used for the generation of Ri -Mp RALF1 mutants

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Anti-musk Abs are thought to block the binding of other components, including collagen Q and low-density lipoprotein receptor-related protein 4 (LRP4) that facilitate the role of MuSK in the neuromuscular junction [149, 150]