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A potential explanation is that GL bound to HLA-C*05:01 is sufficient for KIR2DL1 binding

When a peptide meets its receptor, its like a text message to the cell: Repair here, Make more collagen, or Turn down inflammation. Many therapeutic peptides are synthetic or lab-refined versions of molecules your body already produces, so they tend to work by amplifying or fine-tuning the bodys own signals rather than overriding them

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