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For instance, deleting Pellino3 exacerbates insulin resistance by increasing pro-inflammatory macrophages, while inhibiting ITCH protects against steatohepatitis by promoting anti-inflammatory macrophages

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These analogs allosterically mimic dimerization/activation, a prerequisite to binding to the Type 1 tyrosine kinase receptor c-Met [29,30]

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Oxidative/electrophilic stress sensor Keap1 regulates the rapid turnover of transcripition factor Nrf2

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