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Dysregulated histone acetylation is increasingly linked to tumor progression and therapeutic resistance, where acetylation-driven activation of survival and redox pathways fosters drug toleranceincluding reduced sensitivity to AR-directed therapies in CRPC [25,26,27,28]

Current Diabetes Reviews, 16

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is supported, in part, by a Banting and Best Diabetes CentreNovo Nordisk Chair in Incretin Biology, a Sinai HealthNovo Nordisk Foundation Fund in Regulatory Peptides, CIHR grants 154321 and 19204 and Diabetes Canada-Canadian Cancer Society grant (OG-3- 24-5819-DD)