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Similarly, the same pathological changes have been observed in the BV + DOX-treated group (Fig
69 Furthermore, the overexpression of claudin-1 in the intestinal epithelium caused hyperactivation of Notch signaling and suppressed goblet cell differentiation and mucin-2 synthesis
TPE is performed under the care of a licensed medical provider and monitored by healthcare professionals
Next, we examined the enzymatic activities of AsGGT1, AsGGT2, and AsGGT3 toward -glutamyl- S -allyl-L-cysteine and -glutamyl- S -allyl-L-cysteine sulfoxide, which are two possible biosynthetic intermediates in alliin biosynthesis, as potential -glutamyl donor substrates ( Figures 3 and 4 )
doi: 10.1007/BF03345785 200 HuangZRoseAHHoffmannPR
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