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N-acetyl-cysteine maintains cell health, is also very beneficial for cell metabolism, and supports cells from damage caused by toxins

Chemically induced hepatocyte-like cells (HLCs) are another hepatocyte source for HTx

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11 In cardiac tissues, GLP-1R activation reduces oxidative stress and mitochondrial dysfunction via the cAMP/Epac/PI3K/AKT pathway, with implications for cell survival and stress adaptation

This potential age-related effect may be secondary to a progressive imbalance between ROS production and waning antioxidant defenses during aging, making G6PD-d individuals more susceptible to oxidative stress-induced ASCVD 22

Rahman, 2025)