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Histological analyses revealed partial improvement in pancreatic, hepatic, and renal tissues following treatment with the plant extract (380)
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The mechanistic explanation is not speculative: the glucagon receptor adds hepatic fatty-acid oxidation, adipose lipolysis and thermogenic energy expenditure that dual agonism cannot engage by design 27, and the Phase 2a MASLD readouts 8590% liver-fat reduction is a direct signature of that pathway 3
No pharmacological overlap washout is needed because both drugs are GLP-1 agonists, though re-escalating through the full semaglutide dose ladder is recommended to minimize GI side effects
