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Initial enthusiasm that such mutations accounted for all or most classical EDS has been restrained by subsequent findings, which suggest that 3050% of classical cases are caused by COL5A1 mutations and a substantially smaller number by COL5A2 mutations

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Intracerebroventricular injections of the enteric bacterial metabolic product propionic acid impair cognition and sensorimotor ability in the Long-Evans rat: further development of a rodent model of autism

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