doi: 10.3390/separations10060362 94 Pereira-CoelhoMda Silva HaasICVitaliLdos Santos MadureiraLA
Zhao X, Wang Z, Wang J, Xu F, Zhang Y, Han D, Fang W
(2020) specifically addressed NSAID-induced gut damage a clinically relevant injury model and documented that BPC-157 administration stabilized the claudin-4 and occludin tight junction proteins that are degraded by NSAID exposure, providing a mechanistic explanation for the peptides permeability-protective effects
Thus, severe sodium restriction in animals with early heart disease could theoretically be detrimental by early and excessive activation of the RAA system (Pedersen 1996
Biochemical and biophysical research communications , 280 (1), 132138.
PLoS One 2016;11:e0154037