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Side effects are dose-dependent and typically resolve quickly after application adjustment

Moreover, several metabolites in the plasma of PD patients are significantly altered compared with those in the plasma of healthy controls [251]

When toxic exposure or chronic inflammation is too high, Phase 1 rapidly uses up the liver's glutathione reserves, causing Phase 2 to come to a screeching halt

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An additional connection between PD and glucose metabolism derives from the observation of the role of bromocriptine: a DA agonist active on D2 historically used for PD, which was later approved by Food and Drug Administration to treat T2DM (Pijl et al., 2000)

It may also enhance cognitive function, vitality, and longevity when taken as a daily supplement