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Peptides in solution can form aggregates over time, particularly when exposed to mechanical stress, temperature cycling, or surface contact

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In gene expression profiling, GHK-Cu treatment of human fibroblasts upregulated superoxide dismutase (SOD1, SOD2, SOD3) and glutathione-related genes while suppressing NF-kB-driven inflammatory signaling [3]

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The peptide's anti-inflammatory effects, mucosal healing promotion, and intestinal barrier function enhancement address key pathological features of IBD