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In addition to forming cancer cells, ROS overproduction contributes to cell migration by degrading the extracellular matrix, disrupting cell-to-cell contact, remodeling the cytoskeleton, regulating gene expression, and forming invadopodia [82]

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MeJa treatments were able to maintain cell wall metabolism after storage by upregulating polygalacturonase, pectate lyase, and expansin-related genes

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