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Standard effect sizes (Hedges g) for individual studies were estimated

Previous studies have demonstrated that electroconvulsive shock can activate microglia, upregulate neuroinflammatory cytokines, and enhance OS responses [46, 47]

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Because endothelial cells are subjected to severe oxidative stress in ARDS, this mechanism is especially pertinent

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GHK-Cu represents the "external" face of longevity research skin and connective-tissue aging versus the cellular and metabolic focus of the other three