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In summary, animal models show that Thiazolidinediones may stimulate mitochondrial biogenesis, reduce inflammation, and attenuate ischemiareperfusion lesions in organs

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The results indicate that targeting GPX4 to promote ferroptosis may be an effective technique for treating therapy-resistant malignancies, such as TNBC

BPC-157 has demonstrated healing effects in models of inflammatory bowel disease, gastric ulceration caused by non-steroidal anti-inflammatory drugs, short bowel syndrome, and intestinal fistulas

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This route expanded after the April 2026 reclassification opened the injectable pathway