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Some clinics recommend ongoing therapy for best results, making it a significant long-term expense

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This period is less about maintenance and more about robust support for cellular integrity and immune function

To confirm the binding mode between c-MET II and HGF I as shown in our structure, we introduced three point mutations in the K1 domain of HGF, including E159R, E195R, and R197E, to disrupt this interface, and examined the effects of these mutations on c-MET activation