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Decreased dopamine levels correlated with the development of colitis in both humans and rodents, and rodent studies showed that dopamine signaling through D5-mediated changes in macrophage phenotype that decreased inflammation (Magro et al., 2004

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QA also acts as a competitive agonist at the NMDAR, with selectivity for the GluN2A and GluN2B subunits [34]

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