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Chronic sleep deprivation can lead to lower baseline levels of glutathione, making supplementation even more vital

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Nanoliposomes dual-modified with the brain injury site-targeting peptide CAQK and TPP can deliver 4-octyl itaconate (4-OI) to the mitochondria of cells in the brain, inhibiting the activation of the NLRP3 inflammasome and the opening of the mPTP to alleviate neuroinflammatory responses (150)

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Proteomic identification of nitrated brain proteins in traumatic brain-injured rats treated postinjury with gamma-glutamylcysteine ethyl ester: insights into the role of elevation of glutathione as a potential therapeutic strategy for traumatic brain injury