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Therefore, we hypothesize that the benefits of semaglutide can be extended to patients with these cognitive impairments, but more clinical studies are needed in the future to further confirm this

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[3] [4] The medication works by mimicking naturally occurring incretin hormones that regulate blood glucose levels, enhance insulin secretion, suppress glucagon release, and slow gastric emptying

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As a comparison, the ECL1 of GIPR partially unwound with the presence of three proline residues (P195 ECL1 , P197 ECL1 and P199 ECL1 ), resulting in reduced interactions between ECL1 and tirzepatide compared to that in GLP-1R (Fig