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Moreover, it plays an immune negative regulatory role in chronic hepatitis B virus (HBA) infection by preventing the formation of germinal centers (GCs), reducing the antiviral activity of GC-related immune cells and enhancing the function of immunosuppressive cells [227]

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It's the evidence that's been rigorously tested, clearly communicated, and directly relevant to how healthcare actually works outside the trial setting

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doi:10.1111/j.1464-410x.2007.07205.x