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The binding of IGFs to their receptors activates both MAPK and PI3K-AKT pathways, which results in downstream cellular effects (such as cellular proliferation, anti-apoptosis, and differentiation actions) through other downstream molecules ( IRS 1-4 and other signaling proteins, such as Shc, bind to the IGF-1R at the extracellular subunit as a result of the binding of IGF-I and IGF-II ( Recent research has shown that the IGF-1R (and IR) can migrate to the nucleus in both healthy and cancerous cells through mechanisms that are still being fully elucidated, although sumoylation is thought to be one method

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It is worthy to note that activation of adrenergic receptors may induce massive Ca 2+ release from endoplasmic reticulum which can be important for coordination of cellular responses to stimulation ( 2.5 ROS-related approaches to cope with PTSD In this section we will build on the involvement of mitochondria in enhanced ROS levels and induction of oxidative stress in PTSD, and describe a molecular basis at least for two groups of treatments

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