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10.3389/fnins.2022.985754 Front

15 This review comprehensively introduces the biological role, molecular mechanisms, and progress of ferroptosis in various human diseases

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By simultaneously pushing the accelerator via GHRH receptors and removing the brakes via somatostatin inhibition, the resulting pulse is significantly more potent than either peptide could produce in isolation

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In digesting protein, the GI serves them up to small intestinal epithelia mainly as di- and tripeptide substrates for the PEPT1- and other enterocyte carriers