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It turns down overactive inflammation

In humans, PAGln enhances platelet reactivity and thrombotic potential through ADR signaling, accelerating thrombus formation in arterial injury models while promoting systemic inflammation and endothelial dysfunction to drive atherosclerosis ( and promote M1 macrophage polarization, accelerating atherosclerosis

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Deciding on the right dose should always be done under the guidance of a qualified healthcare professional

Importantly, these compounds did not prevent TGF--induced HSC activation or reduce expression of ECM genes, including -SMA and COL1A1 [39]