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Moreover, calcitriol has been described to influence cell-specific regulatory processes including certain aspects of the NOD-like pattern recognition receptor signaling, along with enhancing the catabolism of branchedchain amino acids (BCAAs) in monocytic cells potentially resulting in the suppression of BCAA-regulated mTOR signaling [147]

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In addition to iron-catalyzed free radical chain reactions, lipoxygenase (LOXs) can directly oxidize PUFAs and lipid-containing PUFAs in cell membranes, which are considered to have an essential role in ferroptosis [75, 80]

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