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Functionally, persistent cardiac mitochondrial dysfunction is consistent with restricted ATP production, that could impair contractility and increase susceptibility to arrhythmogenesisfindings consistent with chest pain, palpitations, and POTS observed clinically (56)

Deviations from steady state glutathione redox homeostasis have been discussed as a possible mean to alter the activity of redox-sensitive proteins through switching of critical thiol residues

Persistent vomiting, inability to keep fluids down, severe abdominal pain, or extreme fatigue are not normal and should be evaluated

T., Xu D., Zhou J