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They manifest as areas of high cellular density that typically blend with the surrounding nevus component

Thus, future studies should include other inflammation-related biomarkers which may explain part of the variance in the clinical neuropsychiatric symptoms, including autoimune responses, the cytokine network, the NLRP3 inflammasome, and hypernitrosylation

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However, research done on rats or mice doesnt always correlate with the research done with human participants

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doi: 10.1074/jbc.274.34.24408 73 OGormanDBCostelloMWeissJFirthSMScottCD