The researchers also conducted RNA-sequencing, which apparently pointed to reduced activity of pathways involved in ECMreceptor interaction and focal adhesion, alongside lower levels of key ECM proteins such as fibronectin, tenascin C, laminin, thrombospondin-2, and Hmmr
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Glucagon acting at the GLP-1 receptor contributes to -cell regeneration induced by glucagon receptor antagonism in diabetic mice
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