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Research showed that glucagon co-agonism increased hepatic fat oxidation and thermogenesis, providing an additional fat-loss mechanism distinct from appetite suppression alone

One hospitalization for unstable angina occurred, and no revascularization procedures were recorded

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Experimental work has focused on receptor pharmacology, cyclic AMP linked signaling, food intake and energy expenditure pathways, hepatic lipid handling, inflammatory markers, and cross-tissue metabolic communication in animal and biochemical models

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