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32,33,35 The lack of accumulation of intermediary metabolites suggests that the defect is located in the initial step of methionine transformation regulated by SAM synthe-tase

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B12 supports red blood cell production and normal energy metabolism, so correcting a deficiency can help recovery

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The thirty (30) cerebral glucose metabolism ROI measures, as well as the clinical measures (SF36, MFIS, MHI, and PDQ), were studied separately using linear mixed effects (LME) models with random effect of patient

Similar findings for Cx43 at the cell membrane have also been reported 44