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J Urol 1979

Mathew B, Amaechi C, Asante K, Albuquerque E, Benedict A, Kneeream E, Lopez L, et al

Patients with OA exhibit elevated iron levels in synovial tissue, and the impairment of iron homeostasis-regulating genes causes cellular iron accumulation, resulting in heightened production of ROS as well as the increased level of collagen II and MMP13, which are established hallmarks of OA [85] (Figure 2)

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