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Riboflavin, in the form of FAD, acts as a cofactor of MTHFR and molecular studies show that the variant enzyme becomes inactive as a result of an increased propensity to dissociate from FAD (Reference Pejchal, Campbell and Guenther109,Reference Yamada, Chen and Rozen110)

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Limitations of the study In this comprehensive evaluation of Semas impact on myocardial remodeling, our investigation employed a dose-response approach, administering 4.0, 12.0, or 60.0 mg/kg/day of Sema